HMGB1/2 acted as a target for corynoxine B (Cory B) to promote

Corynoxine promotes TFEB/TFE3-mediated autophagy and alleviates Aβ pathology in Alzheimer's disease models

Min LI, Hong Kong Baptist University, Kowloon, Research and Development Division, School of Chinese Medicine

HMGB1 is involved in autophagy inhibition caused by SNCA/α-synuclein overexpression: a process modulated by the natural autophagy inducer corynoxine B. - Abstract - Europe PMC

Fe65-engineered neuronal exosomes encapsulating corynoxine-B ameliorate cognition and pathology of Alzheimer's disease

Loss of HMGB1 exacerbates DSS and Il10 –/– colitis. ( A ) Survival

Chris TAN, Associate Professor, Bachelor of Science, Southern University of Science and Technology, Shenzhen, SUSTech, Department of Chemistry

Calpain inhibition rescues DSS colitis. Hmgb1 fl/fl and Vil-Cre Hmgb1

Corynoxine B targets at HMGB1/2 to enhance autophagy for α-synuclein clearance in fly and rodent models of Parkinson's disease - ScienceDirect

HMGB1 is a Potential and Challenging Therapeutic Target for Parkinson's Disease

Corynoxine B derivative CB6 prevents Parkinsonian toxicity in mice by inducing PIK3C3 complex-dependent autophagy

Analysis of research quality of chemical autophagy modulators. (A) The

Corynoxine B targets at HMGB1/2 to enhance autophagy for α-synuclein clearance in fly and rodent models of Parkinson's disease - ScienceDirect

HMGB1 is involved in autophagy inhibition caused by SNCA/α-synuclein overexpression: a process modulated by the natural autophagy inducer corynoxine B. - Abstract - Europe PMC

Jiahong Lu's research works University of Macau, Macau and other places

Frontiers Immunological Significance of HMGB1 Post-Translational Modification and Redox Biology